{"id":12764,"date":"2023-09-14T19:15:40","date_gmt":"2023-09-14T19:15:40","guid":{"rendered":"https:\/\/fadingstar.mx\/?p=12764"},"modified":"2024-02-23T00:38:32","modified_gmt":"2024-02-23T00:38:32","slug":"ros","status":"publish","type":"post","link":"https:\/\/v4.fadingstar.mx\/2023\/09\/14\/ros\/","title":{"rendered":"ROS"},"content":{"rendered":"\n
The premise behind “pep” (post exposure prophylaxis) for “hiv” is that you take it (for 28 days) within 72 hours of a single exposure.<\/p>\n\n\n\n
Presumably this acts on blood stages before they can invade apicoplasts. I’m not sure if this does anything about the apicoplasts – if it doesn’t then it means that “pep” and “hiv diagnosis” are flawed.<\/p>\n\n\n\n
All “hiv” medicines treat blood stages, not apicoplasts. apologies if i have the exact nomenclature for how it infects rbcs and liver stages wrong. but once the blood stages of the parasite get inside of the apicoplasts<\/strong> in an rbc or a liver stage, current meds dont touch it. it stops the “import” it blocks the “protease” it doesnt fix the apicoplast to the best of my current understanding – this part i admit is a little tentative right now and up until today I didnt know that these apicoplasts were in both RBCs and in liver cells. please accept my apologies if prior entries “sound like i didn’t know that.” <\/p>\n\n\n\n Bear in mind when these soldiers came home with p. falciparum in the 1970s we still wouldn’t know what an “apicoplast” even was for several more decades so don’t even give me that “nooooo the science was already settled” shit:<\/p>\n\n\n\n